By David H. Ingbar, Joseph M. Lasnier (auth.), Sadis Matalon, Jacob Lasha Sznajder (eds.)
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Additional info for Acute Respiratory Distress Syndrome: Cellular and Molecular Mechanisms and Clinical Management
Significantly different from normoxic control value. U5 . 39 Na,K-ATPase, the enzyme subunits predominantly expressed in ATII cells,30,3 1 were reduced after at least 6 h of 0% 0 2 exposure (Fig. 4). , <:> 20 U C~ :J '= = t ::'" :c. '" -< I ~ oi Z t50 13 1 1 ~~~ . / ~ tOO 50 0 * " I I ~ *..! ,- and Figure 4. Effect of hypo xia and rcoxygcnation on mRNA expres sion of subunits in ATll ce lls. 6. 12 or 18 h. 0 2). At the end of ex posure. RNase protection assays we re perfo rmed on ccl i lysates (RNA equivale nt to (() 6 cells).
WeiI, Hypoxia-induced increases in pulmonary transvascular proteinescape in rats. 1. Clin. Invest . 82:1840-1847 (1988). 5. G. Basset. C. Crone, and G. Saumon. Significance of active ion transportin transalveolar water absorption : a study on isolated rat lung,J. Physio l. Lond . 384:311-324 (1987). 6. M. A. Matthay and 1. P. Wiener-Kronish, Intact epithelium barrier function is critical for the resolution of alveolar edema in humans. '1111. Rev. Respir. Dis. 142:1250-1257 (1990). 7. S. Matalon, R.
G. Palmerand G. Frindt, Effectsof cell Ca2+ and pH on Na+channelsfrom rat corticalcollecting tubule, Am. J. Physiol .
Acute Respiratory Distress Syndrome: Cellular and Molecular Mechanisms and Clinical Management by David H. Ingbar, Joseph M. Lasnier (auth.), Sadis Matalon, Jacob Lasha Sznajder (eds.)